Mrs. T.'s suspected ischemic stroke stems from a blockage in brain blood flow, causing an acute lack of oxygen and nutrients, leading to cellular dysfunction and potential neuronal death due to energy deprivation and disrupted ion balance (Su et al., 2022). How will pathophysiological changes transition in the subacute phase after diagnosis and initial treatment? In the subacute phase post-diagnosis and initial treatment, reperfusion injury from restored blood flow could trigger oxidative stress, inflammation, and additional tissue damage. Efforts toward tissue repair may begin, potentially reducing initial swelling and inflammation, yet persistent functional deficits might necessitate ongoing rehabilitation efforts (Kuriakose & Xiao, 2020)
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